Which tremor is defined by fine 6-9 Hz oscillations in limbs, worsened by activity, autosomal dominant, and treated with propranolol?

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Multiple Choice

Which tremor is defined by fine 6-9 Hz oscillations in limbs, worsened by activity, autosomal dominant, and treated with propranolol?

Explanation:
The main concept is a hereditary, action-related tremor that responds to beta-blocker therapy. This pattern describes essential tremor: a rhythmic, fine tremor of the limbs that is most noticeable with posture and voluntary movement, typically in the 6–7 Hz range but often cited around 6–9 Hz. It commonly runs in families in an autosomal dominant fashion, so a positive family history fits well. Propranolol is a first-line treatment for reducing the tremor amplitude. So the features—fine tremor in the limbs at roughly 6–9 Hz, worsened by activity, autosomal dominant inheritance, and response to propranolol—line up best with essential tremor. In contrast, myoclonus would be abrupt, shock-like jerks; chorea would be irregular, unpredictable, dance-like movements; akathisia is a subjective restlessness with constant movement, not a rhythmic, task-dedicated tremor.

The main concept is a hereditary, action-related tremor that responds to beta-blocker therapy. This pattern describes essential tremor: a rhythmic, fine tremor of the limbs that is most noticeable with posture and voluntary movement, typically in the 6–7 Hz range but often cited around 6–9 Hz. It commonly runs in families in an autosomal dominant fashion, so a positive family history fits well. Propranolol is a first-line treatment for reducing the tremor amplitude.

So the features—fine tremor in the limbs at roughly 6–9 Hz, worsened by activity, autosomal dominant inheritance, and response to propranolol—line up best with essential tremor. In contrast, myoclonus would be abrupt, shock-like jerks; chorea would be irregular, unpredictable, dance-like movements; akathisia is a subjective restlessness with constant movement, not a rhythmic, task-dedicated tremor.