Which lesion location is associated with alexia without agraphia?

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Multiple Choice

Which lesion location is associated with alexia without agraphia?

Alexia without agraphia arises when the route from visual input to language processing is disrupted, typically by a lesion in the left posterior brain that involves the occipitotemporal region and the posterior corpus callosum (splenium). The left occipitotemporal cortex contains the visual word form area, which is essential for recognizing written words. If the splenium is damaged, visual information from the right visual field can’t be transferred to the left-hemisphere language system, so reading is impaired while writing remains intact. The combination of left posterior corpus callosum involvement with the left inferior temporal gyrus best accounts for this pattern. Lesions in other areas, like the angular gyrus, produce alexia with agraphia, and frontal or motor-area lesions don’t produce pure alexia.

Alexia without agraphia arises when the route from visual input to language processing is disrupted, typically by a lesion in the left posterior brain that involves the occipitotemporal region and the posterior corpus callosum (splenium). The left occipitotemporal cortex contains the visual word form area, which is essential for recognizing written words. If the splenium is damaged, visual information from the right visual field can’t be transferred to the left-hemisphere language system, so reading is impaired while writing remains intact. The combination of left posterior corpus callosum involvement with the left inferior temporal gyrus best accounts for this pattern. Lesions in other areas, like the angular gyrus, produce alexia with agraphia, and frontal or motor-area lesions don’t produce pure alexia.

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